Person

Oliver Hantschel

This person is no longer with EPFL

Related publications (58)

About
Privacy
Disclaimer

Graph Chatbot

Chat with Graph Search

Ask any question about EPFL courses, lectures, exercises, research, news, etc. or try the example questions below.

DISCLAIMER: The Graph Chatbot is not programmed to provide explicit or categorical answers to your questions. Rather, it transforms your questions into API requests that are distributed across the various IT services officially administered by EPFL. Its purpose is solely to collect and recommend relevant references to content that you can explore to help you answer your questions.

Oliver Hantschel

Graph Chatbot

Chat with Graph Search

Tuning SAS-6 architecture with monobodies impairs distinct steps of centriole assembly

Crizotinib acts as ABL1 inhibitor combining ATP-binding with allosteric inhibition and is active against native BCR-ABL1 and its resistance and compound mutants BCR-ABL1(T315I) and BCR-ABL1(T315I-E255K)

Btk SH2-kinase interface is critical for allosteric kinase activation and its targeting inhibits B-cell neoplasms

Selective inhibition of STAT3 signaling using monobodies targeting the coiled-coil and N-terminal domains

Monobodies as enabling tools for structural and mechanistic biology

γ-Catenin-Dependent Signals Maintain BCR-ABL1+ B Cell Acute Lymphoblastic Leukemia

Targeted protein degradation through cytosolic delivery of monobody binders using bacterial toxins

Rapid Screen for Tyrosine Kinase Inhibitor Resistance Mutations and Substrate Specificity

Structural and functional dissection of the DH and PH domains of oncogenic Bcr-Abl tyrosine kinase

Selective Targeting of SH2 Domain–Phosphotyrosine Interactions of Src Family Tyrosine Kinases with Monobodies