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Organization of the SH3-SH2 unit in active and inactive forms of the c-Abl tyrosine kinase

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Organization of the SH3-SH2 unit in active and inactive forms of the c-Abl tyrosine kinase

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Allosteric Inhibition of Bcr-Abl Kinase by High Affinity Monobody Inhibitors Directed to the Src Homology 2 (SH2)-Kinase Interface

HRD Motif as the Central Hub of the Signaling Network for Activation Loop Autophosphorylation in Abl Kinase

Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases

Identification and functional characterization of protein kinases that modulate Notch signaling under physiological conditions and in cancer

KAT2A/KAT2B-targeted acetylome reveals a role for PLK4 acetylation in preventing centrosome amplification

Crystal structure of an SH2-kinase construct of c-Abl and effect of the SH2 domain on kinase activity

Functional Perturbation of the Gab2 Multiprotein Complex by High-affinity SH2-binding Monobodies in Chronic Myelogenous Leukemia

Targeting BCR-ABL and JAK2 in Ph+ ALL

The SH2 domain of Abl kinases regulates kinase autophosphorylation by controlling activation loop accessibility

c-Abl phosphorylates α-synuclein and regulates its degradation: implication for α-synuclein clearance and contribution to the pathogenesis of Parkinson's disease