Publication

Gene expression profiling of R6/2 transgenic mice with different CAG repeat lengths reveals genes associated with disease onset and progression in Huntington's disease

Related publications (91)

About
Privacy
Disclaimer

Graph Chatbot

Chat with Graph Search

Ask any question about EPFL courses, lectures, exercises, research, news, etc. or try the example questions below.

DISCLAIMER: The Graph Chatbot is not programmed to provide explicit or categorical answers to your questions. Rather, it transforms your questions into API requests that are distributed across the various IT services officially administered by EPFL. Its purpose is solely to collect and recommend relevant references to content that you can explore to help you answer your questions.

Gene expression profiling of R6/2 transgenic mice with different CAG repeat lengths reveals genes associated with disease onset and progression in Huntington's disease

Graph Chatbot

Chat with Graph Search

The CRL5-SPSB3 ubiquitin ligase targets nuclear cGAS for degradation

Statistical learning quantifies transposable element-mediated cis-regulation

Wide-scale identification of novel/eliminated genes responsible for evolutionary transformations

Coordination of protein synthesis and degradation in mammalian cells

Integrative transcriptome analyses of Transposable Elements (TEs) and genes contribute to a better understanding of colorectal cancer

An integrative approach to elucidate the mechanisms and dynamics of Huntingtin aggregation and inclusion formation in neuronal models of Huntington's Disease

GCN2 adapts protein synthesis to scavenging-dependent growth

N-terminal mutant huntingtin deposition correlates with CAG repeat length and symptom onset, but not neuronal loss in Huntington's disease

Ribo-DT: An automated pipeline for inferring codon dwell times from ribosome profiling data

Prioritization of cell types responsive to biological perturbations in single-cell data with Augur