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MicroRNA 21 is a novel oncogene in Notch1-driven acute lymphoblastic T cell leukemia

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MicroRNA 21 is a novel oncogene in Notch1-driven acute lymphoblastic T cell leukemia

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Notch controls embryonic Schwann cell differentiation, postnatal myelination and adult plasticity

Simultaneous loss of beta- and gamma-catenin does not perturb hematopoiesis or lymphopoiesis

Identification of the Design Principles of Signaling Pathways for Metabolic Engineering

In vivo transformation of mouse conventional CD8{alpha}+ dendritic cells leads to progressive multisystem histiocytosis

Notch signaling is required for exocrine regeneration after acute pancreatitis

Palmitoylation and ubiquitination regulate exit of the Wnt signaling protein LRP6 from the endoplasmic reticulum

Hierarchy of Notch-Delta interactions promoting T cell lineage commitment and maturation

Cells with defective p53-p21-pRb pathway are susceptible to apoptosis induced by p84N5 via caspase-6

Notch1 engagement by Delta-like-1 promotes differentiation of B lymphocytes to antibody-secreting cells

Unique mechanisms of growth regulation and tumor suppression upon Apc inactivation in the pancreas