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Crystal structure of an SH2-kinase construct of c-Abl and effect of the SH2 domain on kinase activity

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Crystal structure of an SH2-kinase construct of c-Abl and effect of the SH2 domain on kinase activity

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Unpaired extracellular cysteine mutations of CSF3R mediate gain or loss of function

CRISPR-Cas9-mediated saturated mutagenesis screen predicts clinical drug resistance with improved accuracy

Allosteric regulation and targeting of Abl SH2-kinase interface and other cytoplasmic tyrosine kinases

Allosteric Inhibition of Bcr-Abl Kinase by High Affinity Monobody Inhibitors Directed to the Src Homology 2 (SH2)-Kinase Interface

Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases

HRD Motif as the Central Hub of the Signaling Network for Activation Loop Autophosphorylation in Abl Kinase

Functional Perturbation of the Gab2 Multiprotein Complex by High-affinity SH2-binding Monobodies in Chronic Myelogenous Leukemia

Targeting BCR-ABL and JAK2 in Ph+ ALL

The SH2 domain of Abl kinases regulates kinase autophosphorylation by controlling activation loop accessibility

NUP214-ABL1 mediated cell proliferation in T-cell acute lymphoblastic leukemia is dependent on the LCK kinase and various interacting proteins