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Use of lentiviral vectors for modelling and treating Parkinson's disease

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Use of lentiviral vectors for modelling and treating Parkinson's disease

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Comparative analyses of Purkinje cell gene expression profiles reveal shared molecular abnormalities in models of different polyglutamine diseases

Phosphorylation of α-synuclein is crucial in compensating for proteasomal dysfunction

Alpha-Synuclein Effects at the ER-to-Golgi Level and Potential Biomarkers in Rat Models of the Early Phase of Parkinson's Disease

Rab1A Over-Expression Prevents Golgi Apparatus Fragmentation and Partially Corrects Motor Deficits in an Alpha-Synuclein Based Rat Model of Parkinson’s Disease

Parkin reinvents itself to regulate fatty acid metabolism by tagging CD36

Genetic Mouse Models of Neurodegenerative Diseases

A Rat Model of Progressive Nigral Neurodegeneration Induced by the Parkinson's Disease-Associated G2019S Mutation in LRRK2

Gene expression profiling of R6/2 transgenic mice with different CAG repeat lengths reveals genes associated with disease onset and progression in Huntington's disease

Dopaminergic Neuronal Loss, Reduced Neurite Complexity and Autophagic Abnormalities in Transgenic Mice Expressing G2019S Mutant LRRK2

Probing the different chaperone activities of the bacterial HSP70-HSP40 system using a thermolabile luciferase substrate