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Role of Protein Kinase C and Munc18-1 in a Presynaptic Form of Plasticity, Post-tetanic Potentiation

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Role of Protein Kinase C and Munc18-1 in a Presynaptic Form of Plasticity, Post-tetanic Potentiation

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An Exclusion Zone for Ca2+ Channels around Docked Vesicles Explains Release Control by Multiple Channels at a CNS Synapse

Network-timing-dependent plasticity

Memory formation and recall in recurrent spiking neural networks

Munc18-1 is a dynamically regulated PKC target during short-term enhancement of transmitter release

Emergence of Connectivity Motifs in Networks of Model Neurons with Short- and Long-Term Plastic Synapses

An Alien Divalent Ion Reveals a Major Role for Ca2+ Buffering in Controlling Slow Transmitter Release

Network Activity and Plasticity

Matched Pre- and Post-Synaptic Changes Underlie Synaptic Plasticity over Long Time Scales

Network Self-Organization Explains the Statistics and Dynamics of Synaptic Connection Strengths in Cortex

Inhibitory synaptic plasticity: spike timing-dependence and putative network function