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Antibodies Trap Tissue Migrating Helminth Larvae and Prevent Tissue Damage by Driving IL-4Rα-Independent Alternative Differentiation of Macrophages

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Antibodies Trap Tissue Migrating Helminth Larvae and Prevent Tissue Damage by Driving IL-4Rα-Independent Alternative Differentiation of Macrophages

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Exploiting Old Pathogens to Create New Therapeutics

Immune Antibodies and Helminth Products Drive CXCR2-Dependent Macrophage-Myofibroblast Crosstalk to Promote Intestinal Repair

Concerted Activity of IgG1 Antibodies and IL-4/IL-25-Dependent Effector Cells Trap Helminth Larvae in the Tissues following Vaccination with Defined Secreted Antigens, Providing Sterile Immunity to Challenge Infection

Antibody-Mediated Trapping of Helminth Larvae Requires CD11b and Fc gamma Receptor I

The Intestinal Microbiota Contributes to the Ability of Helminths to Modulate Allergic Inflammation

TRIM33 switches off Ifnb1 gene transcription during the late phase of macrophage activation

A droplet-based heterogeneous immunoassay for screening single cells secreting antigen-specific antibodies

IL-1β Suppresses Innate IL-25 and IL-33 Production and Maintains Helminth Chronicity

The C-type Lectin Receptors Dectin-1, MR, and SIGNR3 Contribute Both Positively and Negatively to the Macrophage Response to Leishmania infantum

Antibodies and IL-3 support helminth-induced basophil expansion