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PGC-1 alpha activity in nigral dopamine neurons determines vulnerability to alpha-synuclein

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PGC-1 alpha activity in nigral dopamine neurons determines vulnerability to alpha-synuclein

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A Multiscale, Systems-Level, Neuropharmacological Model of Cortico-Basal Ganglia System for Arm Reaching Under Normal, Parkinsonian, and Levodopa Medication Conditions

Pronounced α-synuclein pathology in a seeding-based mouse model is not sufficient to induce mitochondrial respiration deficits in the striatum and amygdala

Nano-imaging trace elements at organelle levels in substantia nigra overexpressing alpha-synuclein to model Parkinson's disease

Safinamide inhibits in vivo glutamate release in a rat model of Parkinson's disease

Mitochondrial gene signature in the prefrontal cortex for differential susceptibility to chronic stress

Dopamine induces soluble alpha-synuclein oligomers and nigrostriatal degeneration

Parkin functionally interacts with PGC-1 proportional to to preserve mitochondria and protect dopaminergic neurons

The effect of the functional interaction between PGC-1α and Parkin on mitochondrial quality control in Parkinson's disease

Elucidating the Role of Site-Specific Nitration of alpha-Synuclein in the Pathogenesis of Parkinson's Disease via Protein Semisynthesis and Mutagenesis