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The cGAS–STING pathway drives type I IFN immunopathology in COVID-19

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The cGAS–STING pathway drives type I IFN immunopathology in COVID-19

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Enteric helminth-induced type I interferon signaling protects against pulmonary virus infection through interaction with the microbiota

Activation of the STING-dependent type I interferon response reduces microglial reactivity and neuroinflammation

Type I Interferon Signaling Prevents IL-1 beta-Driven Lethal Systemic Hyperinflammation during Invasive Bacterial Infection of Soft Tissue

New immunomodulatory roles of lymphatic endothelium and implications for immunotherapy

Genetic engineering of hematopoiesis for targeted IFN-α delivery inhibits breast cancer progression

Mycobacterium tuberculosis Requires Phosphate-Responsive Gene Regulation To Resist Host Immunity

Induction of type I IFNs by intracellular DNA-sensing pathways

Spontaneous phthiocerol dimycocerosate-deficient variants of Mycobacterium tuberculosis are susceptible to gamma interferon-mediated immunity

Expression of the leptin receptor outside of bone marrow-derived cells regulates tuberculosis control and lung macrophage MHC expression

Tumor-targeted interferon-alpha delivery by Tie2-expressing monocytes inhibits tumor growth and metastasis